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Benfotiamine may be useful for the treatment of diabetic retinopathy, neuropathy, and nephropathy however most of the effects attributed to benfotiamine are extrapolated from in vitro and animal studies.
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Despite the encouraging results regarding benfotiamine potential to alleviate diabetic microangiopathy, neuropathy and other oxidative stress-induced pathological conditions, its activities and cellular mechanisms during microglial activation have yet to be elucidated.
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The decreasing rate among benfotiamine treated subjects was not significantly different from the rate in the placebo group difference 0.
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Given the anti-inflammatory and anti-oxidative potency of benfotiamine 33,34,35 , we determined in the present study the molecular mechanism underlying its protective role in lps activated bv-2 microglia.
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High-dose thiamine and benfotiamine s-benzoylthiamine o-monophosphate, bt administration reduces pain in animal models of inflammatory and neuropathic pain.
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